"Don't buy the damn biscuits in the first place you silly greedy moo"
Research using mice showed that the neurons in the brains of those who had a mutated Bdnf gene, failed to send chemical signals to the hypothalamus part of the brain after consuming food, which resulted in the mice eating twice as much food as their counterparts whose Bdnf genes were working normally. In humans the Bdnf gene develops whilst in the womb.
A dodgy Bdnf gene thus transmuting into a "gluttony" gene - in my own personal case is quite feasible because firstly even when I was a kid, I was always had a gluttonous appetite and secondly, when ever I overeat, mostly every day, afterwards when I feel a little bloated, I often realise that I've literally eaten roughly twice as much as I really needed !!!
The good news for pharmaceutical companies in the discovery of a "gluttony" gene, is that they can develop anti obesity drugs to combat the faulty Bdnf gene, possibly in collusion with the processed junk food industry who may be chemically manipulating foods so the human brain doesn't know when to stop munching, crunching and dunking...